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Defining Response and Resistance to PI3K and AR inhibition in Prostate Cancer

Brett Stewart Carver

0 Collaborator(s)

Funding source

National Institutes of Health (NIH)

Related projects

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The Crosstalk Between the PI3K/Akt signal and Skp2 in Prostate Cancer Progression

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Exploiting the Genomic Programs Underlying AR-Independent Prostate Cancer

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Imputation-Based Approach to Identify Low Frequency Variants in Prostate Cancer

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Role of HSP90 Family Chaperone Proteins in Cellular Signal Transduction

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